Advances in Immunology, Vol. 33 by Henry G. Kunkel, Frank J. Dixon (Eds.) PDF

By Henry G. Kunkel, Frank J. Dixon (Eds.)

ISBN-10: 012022433X

ISBN-13: 9780120224333

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1980). , 1980). 3%), or lymph node cells than their normal F, female littermates. B. , 1975). , 1980). , 1980). Although the ratio of p vs 6 radioactivity in the defectives resembled that observed in the spleens of neonatal normal mice, it was not clear if this similarity was due to a high incidence of p+6- cells in the immune-defective mice. , 1976a, 1980), which can quantitatively determine the amount of fluorescence on individual lymphoid cells. , 1976a). Thus, whereas approximately 24% of the surface IgM-bearing cells in normal adult mice bore relatively high densities of surface IgM, approximately 63% of surface IgM-bearing cells from adult CBNN mice had this characteristic (Fig.

Macrophage LAF production is known to be induced by LPS through its direct effect on macrophages. When thioglycollate-induced macrophages derived from (CBA/N x DBA/ 2)F, male and female mice were tested for their ability to produce LAF at different concentrations of LPS, they were shown to be equivalent. , 1977), suggest that, at least with regard to these functions, their macrophages are normal. , 1980~). Because of the known importance of antigen-presenting accessory cells in the induction of both TD and TI-2 responses, it was possible that abnormal macrophage function could be responsible for the low/absent responses of immune-defective mice to these antigens.

MINOR LYMPHOCYTE-STIMULATING In mice, the MLR is under the independent control of two genetic systems, the multigenic major histocompatibility complex (Dutton, 1966)and the unigenic non-H-2 Mls locus (Festenstein, 1973). A number of studies have demonstrated that Mls-determined markers are present on B but no T cells. In the first of these studies, von Boehmer and Sprent (1974) demonstrated that thymus cells were not stimulatory, whereas spleen cells and B cells isolated by electrophoresis from thoracic duct lymphocytes were stimulatory.

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Advances in Immunology, Vol. 33 by Henry G. Kunkel, Frank J. Dixon (Eds.)

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