By Irma Pereira MT (ASCP) SH, Tracy I. George MD, Daniel A. Arber MD
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Extra info for Atlas of Peripheral Blood: The Primary Diagnostic Tool
4). The disease may be idiopathic or related to other conditions such as lymphoma or autoimmune disorders, particularly systemic lupus erythematosus. Drugs such as penicillin, 6-mercaptopurine, ribavirin, and a-methyldopa may also promote warm autoantibody production, a process known as druginduced immune hemolysis. 1 Mechanism of microangiopathic hemolysis. Depending on where the RBC folds over the fibrin strand, different sizes of fragmented red cells will be present in the circulation. As shown in this example, these could be the small schistocyte or the larger helmet cell.
11). 3 Large three-dimensional RBC aggregates are readily visible at low power and indicate the presence of cold autoagglutinins. in affected neonates and infants who may present with hydrops fetalis, jaundice, splenomegaly, and severe hemolytic anemia requiring transfusions. PK deficiency is distinguished as one of the nonspherocytic hemolytic anemias. Cases with echinocytes, acanthocytes, bite cells, and microspherocytes have all been documented. The reticulocyte count is markedly increased in these patients (Fig.
3. This phenomenon may cause a falsely decreased RBC count with a normal hemoglobin and high mean corpuscular volume (MCV) value on automated hematology analyzers due to the instrument counting clumps of red cells as single cells with increased diameters, and hence a falsely high MCV. Agglutination is reversed by warming the blood to 37°C. Mycoplasma pneumoniae infection or lymphoma may prompt production of cold autoagglutinins. 2. Warm autoagglutinins: This condition is mediated predominately by IgG antibodies.
Atlas of Peripheral Blood: The Primary Diagnostic Tool by Irma Pereira MT (ASCP) SH, Tracy I. George MD, Daniel A. Arber MD